Angina pectoris is a clinical syndrome, is caused by myocardial blood flow to the arteries that decreases, can cause pain because of changes in aerobic metabolism becomes anaerobic, producing lactic acid and stimulate the emergence of pain. Angina pectoris is a state of coronary heart disease patients with complaints of chest pain, caused by myocardial blood flow can not be required to meet the needs oxygen. Angina pectoris occurs because heart muscle is deprived of oxygen due to disruption of arterial blood flow. Angina pectoris is ischemic heart disease caused by a reduced supply of oxygen and reduced blood flow to the myocardium. Interference occurs because of decreased oxygen supply due to coronary atherosclerosis or coronary artery spasm. Coronary atherosclerosis is the process of accumulation of fatty deposits in arteries (atheroma), is caused by factors age, smoking, eating fatty foods, high blood pressure, obesity and other factors. Coronary spasm is a contraction of the coronary arteries that can lead to angina pectoris and heart attacks. Spasm of blood vessels caused by blood is alkaline.
Broadly speaking, angina pectoris were divided into three types:
1. Classical angina pectoris (stable)
Classical angina pectoris (stable) is due to anatomic blockage of coronary atherosclerosis that coronary blood flow to the heart can not meet growing needs. Classical angina pectoris (stable) occurred after physical work activity, the emotions and after meals
2. Variant angina
Variant angina pectoris (Prinzmetal angina) occurs because of coronary vasospasm, and occur when a person is resting
3. Unstable angina pectoris.
Angina pectoris unstable angina pectoris is a combination of classic and variant angina pectoris. Unstable angina pectoris is characterized by increased frequency and duration of angina attacks.